Your privacy, your choice

We use essential cookies to make sure the site can function. We also use optional cookies for advertising, personalisation of content, usage analysis, and social media.

By accepting optional cookies, you consent to the processing of your personal data - including transfers to third parties. Some third parties are outside of the European Economic Area, with varying standards of data protection.

See our privacy policy for more information on the use of your personal data.

for further information and to change your choices.

Skip to main content
Fig. 4 | Alzheimer's Research & Therapy

Fig. 4

From: Clinical phenotypes of Alzheimer’s disease: investigating atrophy patterns and their pathological correlates

Fig. 4

Pathological load of cortical Aβ, pTau, axonal damage and microvascular deterioration in AD phenotypes and controls. Boxplots of immunoreactivity of histological markers Amyloid beta (A) pTau (D) NfL (G) and COLIV (J) as area% load in selected brain regions in control and AD phenotype groups (behavioral, dysexecutive, logopenic and visuospatial). Radar plots showing the distribution of pathology load among control, typical AD, atypical AD (B, E, H, K), and distinct subtype groups (C, F, I, L). * = p ≤ 0.05, ** = p ≤ 0.01, *** = p ≤ 0.001. Hip = hippocampus, ParaHip = parahippocampal gyrus, GFM = middle frontal gyrus, GTM = middle temporal gyrus, GPS = superior parietal gyrus, Precun = precuneus, PCC = posterior cingulate cortex, OC = occipital cortex

Back to article page